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For example, •O 2 – triggers the intracellular p38 MAPK and JNK pathways, promoting apoptosis while also generating other ROS (such as H 2 O 2 ) to further amplify oxidative damage [218]
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In this situation, we explain that there may be the following three reasons: First and foremost is the current biological state of the injured tissue, including the type of tendon disorders, the stage of tendon healing and the inflammation status
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