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These modifications are functionally interdependent because they rely on accessibility of specific structural features—cysteines, signal peptide cleavage sites, and Asn-(not Pro)-Ser/Thr, respectively—by discrete ER machinery (Ellgaard et al., 2016)

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[177] developed for the first time a novel gut‐targeted controlled‐release H 2 MgH 2 microcapsule, MgH 2 @EC@ES, and evaluated the therapeutic efficacy of the microcapsule in a mouse model of ulcerative colitis, which showed that the microcapsule could dose dependently repair the IBD damage caused by DSS and repair the impaired mitochondrial energy metabolism function in colitis and that a high dose of MgH 2 @EC@ES was more effective than the first‐line drug 5‐aminosalicylic acid, however, the authors did not evaluate the effect of the combination of the two in the article [177] (Table 3)