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Its defining trait is a multi-pathway design, in which each part addresses a different research focus, ranging from matrix and inflammation to repair and cell movement

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These isoforms feature a C-terminal sequence (SLTRKD) that replace the pro-angiogenic CDKPRR sequence in VEGF-Axxx isoforms, 76 allowing them to bind VEGFR2 competitively without initiating complete receptor phosphorylation and signaling, which reduce angiogenic responses