nadph glutathione u87 cells

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Frontiers

Frontiers

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Targeting GSS-mediated glutathione synthesis: 6-Met induces oxidative stress dependent apoptosis via triggering mitochondrial bioenergetics metabolism crisis and mTOR suppression in glioblastoma

Targeting GSS-mediated glutathione synthesis: 6-Met induces oxidative stress dependent apoptosis via triggering mitochondrial bioenergetics metabolism crisis and mTOR suppression in glioblastoma

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Glutathione: Role in Oxidative/Nitrosative Stress, Antioxidant Defense, and Treatments - Raj Rai - 2021 - ChemistrySelect

Glutathione: Role in Oxidative/Nitrosative Stress, Antioxidant Defense, and Treatments - Raj Rai - 2021 - ChemistrySelect

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Ferroptosis as a therapeutic target in glioblastoma: Mechanisms and emerging strategies

Ferroptosis as a therapeutic target in glioblastoma: Mechanisms and emerging strategies

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Tumor-targeted glutathione oxidation catalysis with ruthenium nanoreactors against hypoxic osteosarcoma

Tumor-targeted glutathione oxidation catalysis with ruthenium nanoreactors against hypoxic osteosarcoma

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Glutathione is critical for NK cell-mediated immunity: Cell Reports

Glutathione is critical for NK cell-mediated immunity: Cell Reports

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Disruption of NADPH homeostasis by total flavonoids from Adinandra nitida Merr. ex Li leaves triggers ROS-dependent p53 activation leading to apoptosis in non-small cell lung cancer cells

Disruption of NADPH homeostasis by total flavonoids from Adinandra nitida Merr. ex Li leaves triggers ROS-dependent p53 activation leading to apoptosis in non-small cell lung cancer cells

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Frontiers

Frontiers

4.6 / 5 · 15 reviews

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Oxidative Stress and Antioxidants in Glioblastoma: Mechanisms of Action, Therapeutic Effects and Future Directions

Oxidative Stress and Antioxidants in Glioblastoma: Mechanisms of Action, Therapeutic Effects and Future Directions

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Glutathione (GSH) insufficient promotes the occurrence of ferroptosis

Glutathione (GSH) insufficient promotes the occurrence of ferroptosis

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A mitotic NADPH upsurge promotes chromosome segregation and tumour progression in aneuploid cancer cells

A mitotic NADPH upsurge promotes chromosome segregation and tumour progression in aneuploid cancer cells

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Cellular mechanisms controlling oxidative stress. NADPH is an essential

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Glutathione-Dependent Pathways in Cancer Cells

Glutathione-Dependent Pathways in Cancer Cells

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Pro-Oxidant Auranofin and Glutathione-Depleting Combination Unveils Synergistic Lethality in Glioblastoma Cells with Aberrant Epidermal Growth Factor Receptor Expression

Pro-Oxidant Auranofin and Glutathione-Depleting Combination Unveils Synergistic Lethality in Glioblastoma Cells with Aberrant Epidermal Growth Factor Receptor Expression

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SLC7A11 as a bridge between ferroptosis and disulfidptosis: a promising target for tumor treatment

SLC7A11 as a bridge between ferroptosis and disulfidptosis: a promising target for tumor treatment

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The Glutamate–Glutathione axis in neuropsychiatric disorders and cancer: From shared mechanisms to non-invasive biomarkers

The Glutamate–Glutathione axis in neuropsychiatric disorders and cancer: From shared mechanisms to non-invasive biomarkers

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Frontiers

Frontiers

4.8 / 5 · 26 reviews

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NADPH and glutathione metabolism. Principal pathways for generating

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Role of Glutathione in Cancer: From Mechanisms to Therapies

Role of Glutathione in Cancer: From Mechanisms to Therapies

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NADPH consumption by L-cystine reduction creates a metabolic vulnerability upon glucose deprivation

NADPH consumption by L-cystine reduction creates a metabolic vulnerability upon glucose deprivation

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